Moderator: ofonorow
davea0511 wrote:... he also used purely oxidized vitamin C ... or "orange crap" as I like to call it. This orange crap is the DHA acid that this video you refer to discusses.
I should also add ... his findings are suspect when he also says that DHA A is only available from him and is difficult to manufacture. Again, nothing could be farther froth the truth. Dump out any bottle of vitamin C onto a plate ... this will work faster if you use a brand that has no bioflavonoids (like rose hips, etc). Then leave it uncovered in the sun for a few days. When it turns yellow-ish orange it will be about 50% DHA, 50% AA.
davea0511 wrote:I also would like some information how he did his calculations. You can do a psuedo test for vitamin C absorption by doing a simple finger stick blood glucose test before and after. I suspect this is the method he is using.
ofonorow wrote:Excellent davea0511. We are interested in your background.
I wonder where in the world davea0511 got the idea that dehydroascorbic acid is yellow or orange? (And the method of producing DHAA he proposes is absolute malarkey).
ofonorow wrote:From Dr. Cathcart (on the IV admin video). The IV solutions of sodium ascorbate turn yellow after mixing, and Dr. C speculated that the color change was due to the breakdown into DHA.
ofonorow wrote:Also, are you talking about the video being malarkey? I don't understand and would appreciate specifics.
ofonorow wrote:If DHA is converted to AA inside the cell, wouldn't the GSH (that must be required) be converted to oxidized GSH?
DHA is ingested in the diet and also formed from oxidative reactions in cells and extracellular fluid. However, vitamin C oxidation is readily reversed by mechanisms that rapidly transport DHA into metabolically competent cells and reduce it there to AA. The resulting AA can be utilized in the same cells or else released to the extracellular fluid. DHA recycling mechanisms may decrease the amount of AA that humans need to ingest. Pathological conditions that inhibit DHA recycling may decrease AA concentrations and thereby impair AA-dependent enzymatic and antioxidant activities.
We have been selling solutions containing high concentrations of DHAA for some years now, to many happy, satisfied, and regularly-returning customers; these solutions are crystal clear and colorless, but eventually turn yellow when stored at room temperature. These solutions remain stable (clear, colorless, and with no measurable loss of AA or DHAA) literally for years when stored at freezer temperatures, and even cool temperatures greatly enhance their stability.
ofonorow wrote:I agree, very interesting. OxC, if we rely on your experience, and oxidized AA is not the cause of the yellow color, then why do you think that some sodium ascorbate powders immediately turn yellow in solution? (And some do not.)
ofonorow wrote:I am now very curious. For what application would you be selling DHAA? What is it used for??
But I would guess that yellow byproducts already exist in powders that create yellow solutions immediately. And that the immediate precursors of yellow byproducts already exist in powders that form clear solutions that turn yellow very rapidly. I assume the presence of these byproducts or precursors would be related to the age and storage conditions of the powder. Edited to add this comment:
ofonorow wrote:Sherry Lewin in her 1976 book Vitamin C: Its Biology and Medical Potential, wrote that DHA is more permeable through cell membranes than AA, (apparently can enter cells without a transporter?)...
ofonorow wrote:but it still escapes me how an...oxidized form of vitamin C can get recharged (reduced) without depleting another antioxidant?
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