Frodo wrote:Thanks for your answer and information, Owen. Great! I think it's really clear. Lp(a) can't be genetic. Native peoples don't know heart attacks. Just as animals. If Lp(a) would be genetic, native peoples should have had it too. And then they should have suffered heart attacks. But I've never heard or read about it. According Linus Pauling and Matthias Rath, it's convincing that Lp(a) isn't' genetic and must be a naturally reaction of our body. A surrogate for the lack of vitamin c. And the opinion of the doctors? They always say "it's genetic", if they don't know. And that is unfortunately quite often. According Andrew Saul and Steve Hickey: "Fire Your Doctor"!
A little confusing...A "thing" can be "genetic" yet it can be easily treated. For example, our GULO defect that causes our vitamin C deficiency. We take supplemental vitamin C to overcome our bad "genetics."
So what the doctors are implying is that there are no known drugs that lower Lp(a). (And this is true as common heart drugs ELEVATE Lp(a).) The question is whether the Pauling Therapy (and/or other nutrients) can affect Lp(a) and the jury is still out (and not studied).
In any case, any reader with heart disease and elevated Lp(a) should realize that Pauling's invention is called "Lp(a) Binding Inhibitors" for a reason. It makes the otherwise very "sticky" Lp(a) unable to adhere to the arterial wall (because the lysine receptor is already filled from lysine in the blood.) Ditto proline. One of the 3 or possibly 4 Pauling/Rath patents for Lp(a) binding inhibitors is for the invention of dropping organs during transplants into a solution of vitamin C (and a lysine analog). The plaque literally melts away.
Moral: According to Linus Pauling, if you have high Lp(a) you need to be taking high doses of vitamin C, lysine and proline to inactivate the Lp(a) in the blood.