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Johnwen wrote:Here’s the English version of Dr. Rath’s article by Paul A Taylor
http://www4.dr-rath-foundation.org/News ... ought.html
Actually taking Aspirin and V-C together reduce your chances of stomach bleeding!
Aspirin blocks the absorption of V-C but why not just take a small dose of V-C with your Aspirin? The a couple hours later take your full dose V-C!
Back to the original question on LP(a) Aspirin actually lowers it!
Here’s the details of this!
http://clinchem.aaccjnls.org/content/48/9/1454
Remember LP(a) is like a fireman it comes into action when there is a problem!
Johnwen wrote:Here’s the English version of Dr. Rath’s article by Paul A Taylor
http://www4.dr-rath-foundation.org/News ... ought.html
Actually taking Aspirin and V-C together reduce your chances of stomach bleeding!
Aspirin blocks the absorption of V-C but why not just take a small dose of V-C with your Aspirin? The a couple hours later take your full dose V-C!
Back to the original question on LP(a) Aspirin actually lowers it!
Here’s the details of this!
http://clinchem.aaccjnls.org/content/48/9/1454
Remember LP(a) is like a fireman it comes into action when there is a problem!
Frodo wrote:Pauling/Rath have shown what's better than aspirin. I'm on PT since January. And had a strong mci in June 2016. In addition to PT, I take Omega-3, OpC, pycnogenol and so on.
guitarplayer007 wrote:Frodo wrote:Pauling/Rath have shown what's better than aspirin. I'm on PT since January. And had a strong mci in June 2016. In addition to PT, I take Omega-3, OpC, pycnogenol and so on.
Were you taking aspirin before PT?
Post Number:#10 Post by Johnwen » Sun Mar 11, 2012 4:47 am
First your levothyroxine I believe should be (point) .175mg which is a pretty steep dose. With having your thyroid still in place. Have they done a tsh test on you lately? I betting it’s way low. This in it’s self can cause numerous problems.
Anyway getting back to your LP(a) your taking 20MG of crestor which is a statin drug intended to lower your cholesterol. By the number’s on your test it has done this in fact although your doc is probably happy with these results which are low, your body is doing what it’s suppose to do and that is trying to get more life giving materials out to the body as a whole.
I think every one has heard about the little kid who every time he see’s a fire he see’s firemen at the fire. So he concludes that fires are caused by the fireman. This is the analogy that represents the statin principal. Ie. If cholesterol present in the artery’s it’s causing the problem not trying to cure the real problem.
Putting a little different spin on this picture let’s say a warehouse in a residential neighborhood catch’s fire and people start calling the fire department. If a full compliment of fire equipment and fireman arrive the calls will stop. But what would happen if one truck and say two or three fireman show up and just spray a little water on the fire and the other side of the burning building is fully involved which is threating the houses around it. You can bet your bottom dollar there will be hundred’s of calls to the fire department for more equipment.
LP(a) is like those calls. They are the precursors to the repair process of the arteries the body releases them in numbers and basically counts them when they return. They attach to dead tissue and either stick at the location of problem and attach the other patching materials or attach to the loose ones and carry them out of the body. If the numbers returning are low it means their sticking somewhere in the system and the body raises their levels and sends more patching material out. If the patching material is lacking Like from taking STATINS. The body will still add more LP(a).
We know PT helps heal the artery’s but healing takes time patching is quick. LP(a) goes up when there is a problem even higher when it don’t have the material to do it’s job.
Bottom Line: Taking STATINS causes your LP(a) to rise NOT PT!!!
Post Number:#4 Post by Johnwen » Sun Mar 06, 2011 10:14 pm
What we know! The liver produces LP(a) a very sticky substance in response to damage done somewhere within the vascular walls. LP(a) has a great affinity to L-Lysine meaning it will attach to L-Lysine before it attaches to anything else. LP(a) is the first line glue that starts the cascade of events that leads to plaque build up or can be referred to as bandaging a injury of the vascular system. Dr. Pauling has taught us that L-Lysine causes LP(a) to lose it’s stickiness by giving it the equivalent of a Teflon coat which is then returned to the liver for out processing and not regeneration because it has been rendered useless by the L-Lysine attachment.
To your question, if starting PT which contains a good dose of L-Lysine can it cause a spike in LP(a)? It’s my belief that it would for the following reasons.
The Liver produces LP(a) in response to a signal received by something going on in the stream. If LP(a) is being kept from doing it’s job and the signals are still being received by the liver, it will produce a excess amount till the signal is stopped by the healing of the damage. Thank you Vitamin C! Now the liver cannot compare or ID the returning LP(a) nor reprocess it because of it’s attachment to the L-Lysine making it a useless lipid. This would give the liver the impression the damage needs more since none are returning. This demand and production will continue until the signal has subsided. This is important! Dr. Pauling said “Don’t stop taking his therapy
y! Not even for a single day!!” I think you can see why he said this if you take the lysine out of the equation the excess LP(a) would collect on the damaged area before the Therapy could heal it. Leaving it a progressing glob of goo! For lack of a better description. Then you would have to work twice as hard to clean it up IF POSSIBLE.
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